1. Helicobacter pylori infection is the main causative factor in stomach cancer.

2. Active is loosely equivalent to “acute” and often indicates infection by Helicobacter pylori

3. Helicobacter pylori infection is an essential risk factor in 65–80% of gastric cancers, but only 2% of people with Helicobacter infections develop stomach cancer.

4. Aim To reveal the virulence variation related to vacuolating cytotoxicity of coccoid Helicobacter pylori .

5. 20 Bismuth has also become popular in recent years as a treatment in peptic ulcer to eradicate Helicobacter pylori.

6. Values were too low to judge whether there was any effect associated with Helicobacter pylori colonisation in controls.

7. 17 In conclusion, we investigated the influence of Helicobacter pylori colonisation on gastric mucosal eicosanoid synthesis in patients taking NSAIDs.

8. FeON-FeOFF: the Helicobacter pylori Fur regulator Commutates iron-responsive transcription by discriminative readout of opposed DNA grooves Nucleic Acids Res

9. These differences persisted when subgroups of patients were analysed according to Helicobacter pylori colonisation or degree of mucosal injury.

10. Conclusion: The results revealed significant anti-Helicobacter pylori activity of medicinal plants which could be the potential source of new Bactericidal agents.

11. Abdominal distension also occurs in individuals with lactose intolerance, bacterial food poisoning, parasitic infections like giardia, bacteria like helicobacter pylori (H

12. 22 Helicobacter pylori is recognised as a significant cause of chronic antral gastritis and important in the aetiology of peptic ulceration.

13. Growth-inhibiting, Bactericidal, and urease inhibitory effects of Paeonia lactiflora root constituents and related compounds on antibiotic-susceptible and -resistant strains of Helicobacter pylori.

14. Intestinal absorption of Levothyroxin is around 80%, but only in acid environment and is significantly decreased by proton-pump inhibitors, antacids, atrophic gastritis and Helicobacter pylori infection.

15. However, the effect and mechanism of Capsaicin that modulate the induction of pro-inflammatory cytokine in Helicobacter pylori-infected epithelial cells have not been investigated previously.

16. Background: Whether Concomitant therapy is superior to bismuth quadruple therapy or 14-day triple therapy for the first-line treatment of Helicobacter pylori infection remains poorly understood

17. It is either caused by an autoimmune process which leads to achlorhydria and severe intrinsic-factor deficiency ("classical pernicious anemia") or by atrophic gastritis from other causes, in particular Helicobacter pylori infection.

18. Adding to that evidence , a new study from MIT shows that treating male mice with estrogen dramatically lowers their rates of stomach cancer - specifically , cancers caused by chronic infection by the bacterium Helicobacter pylori .

19. People take Bismuth salts by mouth for inflammation of the lining of the colon (), constipation, diarrhea, indigestion, Helicobacter pylori infection, odor caused by an opening in the belly wall during surgery (ileostomy odor), stomach problems caused by nonsteroidal

20. These patterns tend to stay stable over time and most patients had the same strain of Helicobacter in the stomach and duodenum.

21. Francesca Agriesti, Davide Roncarati, Francesco Musiani, Cristian Del Campo, Mario Iurlaro, Francesca Sparla, Stefano Ciurli, Alberto Danielli, Vincenzo Scarlato, FeON-FeOFF: the Helicobacter pylori Fur regulator Commutates iron-responsive transcription by discriminative readout of opposed DNA grooves, Nucleic Acids Research, Volume 42, Issue 5, 1 March 2014, Pages …

22. Cyclopropane fatty acids (CFAs) are synthetized by the addition of a methylene group from S-adenosyl-l-methionine across the carbon-carbon double bonds of unsaturated fatty acid chains of membrane phospholipids.This fatty acid cyclopropanation, catalyzed by the CFA synthase (CfaS) enzyme, occurs in many bacteria, including the human pathogen Helicobacter pylori Although the Cyclopropane

23. Moving into the pylorus.

24. Gastric cancer is one of the most commonly diagnosed cancers, and in both sexes combined the third leading cause of cancer-related deaths worldwide [1, 2].Prominent differences in age-standardized incidence are observed in different parts of the world ranging from 32.1% in eastern Asia to 8,2% in Western Europe [].Among sporadic (distal) gastric Adenocarcinomas Helicobacter pylori …

25. The pyloric Antrum is the initial portion of the pylorus

26. Achalasia of the pylorus was confirmed by delayed gastric emptying time.

27. Among the conditions treated are dry mouth syndrome, verruciform xanthoma, achlorhydria, mucous cysts, oral submucous fibrosis, oral nevi, cancer of the oral mucosa, maloplakia of the genito-urinary tract, vulvovaginitis, helicobacter plylori infection, duodenal ulcers, peptic ulcers, conditions affecting the uterus and appendicitis.

28. ‘Clusters of corkscrew-shaped, Bacilliform organisms, consistent with Helicobacter heilmannii, were seen in these gastric pits (Figure 2, hematoxylin-eo, original magnification × 100).’ ‘All colonies were composed of Bacilliform cells and showed the ability to utilize methanol as a sole carbon source.’

29. Tyramine was ineffective on the antrum and the duodenum, but contracted the pylorus.

30. H pylori infection was a strong predictor of ulcer recurrences.

31. Resection of digestive sphincters like the pylorus (antrum) or the cardia (terminal esophagus) means reflux.

32. Ery increased motihty index (MI) 10-41 fold in antrum, pylorus and upper small intestine.

33. H pylori was identified by culture and histology as previously described.

34. H. pylori lives only in the slimy, acidic mucus inside our stomachs.

35. H. pylori infection speeds up that cancer progression , to about seven months .

36. Eradication of H. pylori is an effective optional treatment for functional dyspepsia.

37. Results Eighty three patients with H pylori positive gastric ulcers entered the study.

38. Increased basal serum gastrin is related to atrophy and to infection with H pylori.

39. These findings support the hypothesis that H pylori colonisation is directly involved in the hypergastrinaemia.

40. Pylori) is a very common — and yes, Contagious — type of bacteria that infects the digestive tract

41. The H pylori negative and positive healthy volunteers were similar for both basal and peak acid output.

42. Mechanical (strain gauges) and electrical (platin electrodes) activity was chronically examined in the dog stomach, pylorus and small intestine during first postoperative days.

43. 25 Only a minority of patients infected with H pylori will eventually develop a duodenal ulcer.

44. Seven of the parents were positive for H pylori on urease testing, histology, and on culture.

45. 28 There are few data concerning the role of anti- H pylori treatment in gastric ulcer disease.

46. The dynamics and importance of H pylori reduced altered mucosal peptic activity is far from being clarified.

47. It is based upon the ability of H. pylori to convert urea to ammonia and carbon dioxide.

48. After eradication of H pylori, duodenal ulcers do not usually recur and the associated chronic gastritis gradually disappears.Sentencedict.com

49. Atrophy, which is closely related to H pylori infection, is associated with a decline in acid secretion.

50. Omeprazole monotherapy merely suppressed bacterial colonisation, especially in the antral region, and eradicated H pylori in individual cases only.

51. In addition, H pylori eradication speeds up ulcer healing and is associated with healing of previously refractory ulcers.

52. Patients classified as having H pylori infection received an eradication regimen; 107 were reassessed 4 weeks after therapy.

53. 14 The high rate of H pylori ammonia production in uraemic patients should accentuate any ammonia induced effects.

54. Examples of Cramping include menstrual cramps and spasms of the circular muscles of the bowel (irritable colon), blood vessels (vasospasm), and pylorus of the

55. 21 One of the H pylori negative patients was on maintenance haemodialysis and eight were on continuous ambulatory peritoneal dialysis.

56. In healthy men without atrophy, gastric acid secretion is preserved with ageing and is independent of H pylori status.

57. 13 On logistic regression, the presence and extent of gastric metaplasia was not significantly associated with H pylori infection.

58. Of the nine H pylori negative uraemic patients, two had oesophagitis and one had scattered petechiae in stomach and duodenum.

59. 10 On logistic regression,(www.Sentencedict.com) the presence and extent of gastric metaplasia was not significantly associated with H pylori infection.

60. 15 H pylori positive gastritis, and the combination of active duodenitis and gastric metaplasia were independent predictors of duodenal ulceration.

61. Cyclical changes in H pylori infection may cause the variations in basal acid secretion that are seen in duodenal ulcer disease.

62. After eradication of H pylori in the duodenal ulcer patients both their basal acid output and basal gastrin fell by 50%.

63. 16 Thus H pylori infection combined with active ulceration may cause the increased basal acid secretion seen in duodenal ulcer disease.

64. 25 Most investigators have observed that H pylori infection causes a greater percentage increase in the postprandial gastrin than fasting gastrin.

65. By electron microscopy, the apical appearance of amorphous granules can be observed in undifferentiated cells, starting at the pylorus, then at the cardia and finally in the fundus region.

66. The studies were designed to evaluate the H pylori eradication potency of the various regimens and the post-therapeutic course of ulcer disease.

67. 22 In conclusion, our findings suggest that H pylori infection may have been predominantly acquired at a young age in the past.

68. 18 The primary aim of the second trial was to see if eradication of H pylori led to resolution of intestinal metaplasia.

69. 8 It has also been proposed that H pylori ammonia production will lead to mucosal damage by denaturing the protective mucus layer.

70. Discussion Our current results confirm that treatment which both heals duodenal ulcers and eradicates H pylori significantly decreases basal plasma gastrin concentrations.

71. 30 The studies were designed to evaluate the H pylori eradication potency of the various regimens and the post-therapeutic course of ulcer disease.

72. And recently, we used to think that stomach ulcers were just caused by stress, until we discovered that H. pylori was the culprit.

73. 16 The role of the increased serum gastrin concentration induced by H pylori in the pathogenesis of duodenal ulcer disease is also unknown.

74. 24 The role of the increased serum gastrin concentration induced by H pylori in the pathogenesis of duodenal ulcer disease is also unknown.

75. 16 The finding of enhanced fasting gastrin concentrations in H pylori positive subjects and in duodenal ulcer disease can not easily be explained.

76. At age 24 weeks , 16 weeks after being infected with H. pylori , male mice in this study were treated with estrogen , Tamoxifen , both or neither .

77. Pylori-related gastritis in the Antrum strongly correlates with an increased risk of peptic ulcer disease, and of duodenal ulcer in particular (the duodenal ulcer phenotype of gastritis)

78. We aimed to compare the efficacy and safety of 10-day Concomitant therapy, 10-day bismuth quadruple therapy, and 14-day triple therapy in the first-line treatment of H pylori.

79. In the group of the 722 patients with nonadvanced disease, achalasia of the pylorus was present in 140 (19.4%), and in the total of 840 patients, uncomplicated cholelithiasis without chagasic involvement of the gallbladder and/or papilla was observed in 58 (6.9%).

80. Three surgical complications of Chagas' disease—megaesophagus, achalasia of the pylorus, and cholelithiasis—were evaluated within the framework of the experience acquired in the management of 840 cases of megaesophagus—722 in the nonadvanced stage of the disease and 118 with advanced disease (dolichomegaesophagus).